Peripheral mononuclear leukocyte DNA damage, plasma prolidase activity, and oxidative status in patients with benign prostatic hyperplasia


Creative Commons License

Gecit I., Meral I. , Aslan M., Kocyigit A. , Celik H., Taskin A., ...More

REDOX REPORT, vol.20, pp.163-169, 2015 (Journal Indexed in SCI) identifier identifier identifier

  • Publication Type: Article / Article
  • Volume: 20
  • Publication Date: 2015
  • Doi Number: 10.1179/1351000214y.0000000121
  • Title of Journal : REDOX REPORT
  • Page Numbers: pp.163-169
  • Keywords: Benign prostatic hyperplasia, Prolidase, Peripheral leukocyte DNA damage, Total antioxidant capacity, Total oxidant status, Oxidative stress index, ANTIOXIDANT ENZYME-ACTIVITIES, CANCER, DISEASE, STRESS, PREVENTION, COLLAGEN, ASSAY, PATHOGENESIS, MATRIX, CELLS

Abstract

Objectives: Prolidase plays a major role in collagen turnover, matrix remodeling, and cell growth. Benign prostatic hyperplasia (BPH) may be associated with an increased extracellular matrix deposition. Therefore, the present study was designed to investigate the plasma prolidase activity, oxidative status, and peripheral mononuclear leukocyte DNA damage in patients with BPH.